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CURRENT PUBLICATIONS
PTP{sigma} Is a Receptor for Chondroitin Sulfate Proteoglycan, an Inhibitor of Neural Regeneration
This study shows that a transmembrane protein tyrosine phosphatase, PTP{sigma}, binds with high affinity to neural chondroitin sulfate proteoglycans. [Science]
Induced Spreading Depression Evokes Cell Division of Astrocytes in the Subpial Zone, Generating Neural Precursor-Like Cells and New Immature Neurons in the Adult Cerebral Cortex
Using neural progenitors isolated in the cortex, researchers set out to determine whether a subgroup of mature cells in the adult cortex have the potential to divide and generate neural precursors. [Stroke]
Bilateral Interaction Between Cord Blood-Derived Human Neural Stem Cells and Organotypic Rat Hippocampal Culture
As a simplified alternative to in vivo transplantation in this work the researchers have applied a long-term organotypic rat hippocampal slice culture as a recipient tissue to study bilateral graft/host cells interactions ex vivo. [Stem Cells Dev]
Regulated Expression of the Ras Effector Rin1 in Forebrain Neurons
The data presented provides insight into regulated expression of a neuronal Ras effector, defines a promoter useful in telencephalic neuron studies, and describes a novel REST site variant directing expression to mature neurons. [Mol Cell Neurosci]
Differential Patterning of Neuronal, Glial and Neural Progenitor Cells on Phosphorus-Doped and UV Irradiated Diamond-Like Carbon
In the present study the researchers complement and extend the findings that phosphorus-doped DLC could be used to produce patterned neuron networks, by exploring patterning of dorsal root ganglion explants, human neural progenitor cells and U-87 astroglioma cells on P:DLC. [Biomaterials]
Tumor Necrosis Factor-{alpha} Impairs Neuronal Differentiation but not Proliferation of Hippocampal Neural Precursor Cells: Role of Hes1
This paper indicates that exposure of hippocampal NPCs to TNF{alpha} when they are undergoing differentiation but not proliferation has a detrimental effect on their neuronal lineage fate, which may be mediated through increased expression of Hes1. [Mol Cell Neurosci]
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